Elevated luteinizing hormone contributes to atherosclerosis formation by inhibiting nitric oxide synthesis via PI3K/Akt pathway.
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DOI: 10.1016/j.vph.2019.106582
Abstract: BACKGROUND The contentious effects of estrogen therapy on the risk of postmenopausal cardiovascular disease (CVD) indicate that this type of atherosclerosis is not solely induced by estrogen deficiency. Other sex hormones such as elevated luteinizing… read more here.
Keywords: pi3k; elevated luteinizing; luteinizing hormone; female mice ... See more keywords