Tenascin-X Mediates Flow-Induced Suppression of EndMT and Atherosclerosis
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DOI: 10.1161/circresaha.121.320694
Abstract: Background: Endothelial-to-mesenchymal transition (EndMT) has been identified as a critical driver of vascular inflammation and atherosclerosis, and TGF-β (transforming growth factor β) is a key mediator of EndMT. Both EndMT and atherosclerosis are promoted by… read more here.
Keywords: endmt; endmt atherosclerosis; flow; tgf ... See more keywords