The neuroendocrine effects of dehydroepiandrosterone and 17β-estradiol in the in vitro preterm hyperoxia infant model.
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DOI: 10.31083/j.jin.2021.01.333
Abstract: Preterm birth causes neurological deficits. Previously, we demonstrated that fetal zone steroids reduce hyperoxia-mediated cell death in vitro. In immature oligodendrocytes (OLN-93 cells), dehydroepiandrosterone + 17β-estradiol co-treatment had synergistic beneficial effects while signals were transduced… read more here.
Keywords: estradiol dehydroepiandrosterone; oln cells; hyperoxia; treatment ... See more keywords