NADPH oxidase 2 deletion enhances neurogenesis following traumatic brain injury
Sign Up to like & getrecommendations! Published in 2018 at "Free Radical Biology and Medicine"
DOI: 10.1016/j.freeradbiomed.2018.05.069
Abstract: Abstract The NADPH oxidase (NOX) enzyme family is a major source of reactive oxygen species (ROS) and contributor to the secondary pathology underlying traumatic brain injury (TBI). However, little is known about how NOX‐derived ROS… read more here.
Keywords: deletion; brain; following tbi; brain injury ... See more keywords
The MAKRO Screening - an assessment tool for discourse deficits in adults with dysexecutive symptoms following TBI.
Sign Up to like & getrecommendations! Published in 2022 at "Brain injury"
DOI: 10.1080/02699052.2022.2034957
Abstract: INTRODUCTION Discourse impairments are common sequelae following TBI. Even though handling discourse is thought to be a basic requirement for social participation and quality of life, few test procedures to assess discourse disorders have been… read more here.
Keywords: assessment tool; following tbi; tool discourse; discourse ... See more keywords
Beta1-receptor blockade attenuates atherosclerosis progression following traumatic brain injury in apolipoprotein E deficient mice
Sign Up to like & getrecommendations! Published in 2023 at "PLOS ONE"
DOI: 10.1371/journal.pone.0285499
Abstract: Traumatic brain injury (TBI) is associated with cardiovascular mortality in humans. Enhanced sympathetic activity following TBI may contribute to accelerated atherosclerosis. The effect of beta1-adrenergic receptor blockade on atherosclerosis progression induced by TBI was studied… read more here.
Keywords: traumatic brain; following tbi; atherosclerosis; brain injury ... See more keywords