Inhibition of glial hemichannels by boldine treatment reduces neuronal suffering in a murine model of Alzheimer's disease
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DOI: 10.1002/glia.23182
Abstract: The contribution of reactive gliosis to the pathological phenotype of Alzheimer's disease (AD) opened the way for therapeutic strategies targeting glial cells instead of neurons. In such context, connexin hemichannels were proposed recently as potential… read more here.
Keywords: boldine treatment; alzheimer disease; glial hemichannels; neuronal suffering ... See more keywords