Histone H4R3 symmetric di-methylation by Prmt5 protects against cardiac hypertrophy via regulation of Filip1L/β-catenin.
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DOI: 10.1016/j.phrs.2020.105104
Abstract: BACKGROUND AND PURPOSE Although histone lysine methylation has been extensively studied for their participation in pathological cardiac hypertrophy, the potential regulatory role of histone arginine methylation remains to be elucidated. The present study focused on… read more here.
Keywords: cardiac hypertrophy; methylation; h4r3me2s; prmt5 ... See more keywords