New thiazolidinones reduce iron overload in mouse models of hereditary hemochromatosis and β-thalassemia
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DOI: 10.3324/haematol.2018.209874
Abstract: Genetic iron-overload disorders, mainly hereditary hemochromatosis and untransfused β-thalassemia, affect a large population worldwide. The primary etiology of iron overload in these diseases is insufficient production of hepcidin by the liver, leading to excessive intestinal… read more here.
Keywords: iron; hemochromatosis thalassemia; hereditary hemochromatosis; iron overload ... See more keywords