mTOR Modulation of IKr through hERG1b-Dependent Mechanisms in Lipotoxic Heart
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DOI: 10.3390/ijms23158061
Abstract: In the atria, the rapid delayed rectifier channel (IKr) is a critical contributor to repolarization. In lipotoxic atria, increased activity of the serine/threonine mammalian target of rapamycin (mTOR) may remodel IKr and predispose patients to… read more here.
Keywords: herg1b; mtor modulation; modulation ikr; ikr ... See more keywords