Deficiency of tenascin-C attenuated cardiac injury by inactivating TLR4/NLRP3/caspase-1 pathway after myocardial infarction.
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DOI: 10.1016/j.cellsig.2021.110084
Abstract: Inflammation and pyroptosis play a deleterious role in cardiac dysfunction after myocardial infarction (MI). NLRP3/caspase-1 is a well-established axis in pyroptosis and inflammation. In this study, we examined the effects of TN-C on pyroptosis through… read more here.
Keywords: caspase; inactivating tlr4; nlrp3 caspase; pyroptosis ... See more keywords