Suppression of NADPH oxidase attenuates hypoxia-induced dysfunctions of endothelial progenitor cells.
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DOI: 10.1016/j.bbrc.2016.11.161
Abstract: NADPH oxidases (NOX) - derived reactive oxygen species (ROS) contribute to oxidative injury in hypoxia-induced pulmonary arterial hypertension. This study aims to evaluate the status of NOX in endothelial progenitor cells (EPCs) under hypoxic condition… read more here.
Keywords: induced dysfunctions; suppression; progenitor cells; endothelial progenitor ... See more keywords