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Published in 2019 at "Neuroscience Letters"
DOI: 10.1016/j.neulet.2019.134301
Abstract: We previously demonstrated that sodium channel 1.7 (Nav1.7) in trigeminal ganglion (TG) was a critical factor in temporomandibular joint (TMJ) inflammation-induced hypernociception, but the mechanism underlying inflammation-induced upregulation of Nav1.7 remained unclear. Glial-neuron interaction plays…
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Keywords:
induced hypernociception;
hypernociception;
sodium channel;
inflammation ... See more keywords