Inhibition of glycolysis disrupts cellular antioxidant defense and sensitizes HepG2 cells to doxorubicin treatment
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DOI: 10.1002/2211-5463.12628
Abstract: Increased glucose consumption is a known hallmark of cancer cells. Increased glycolysis provides ATP, reducing agents and substrates for macromolecular synthesis in intensely dividing cells. Therefore, inhibition of glycolysis is one strategy in anticancer therapy… read more here.
Keywords: inhibition glycolysis; glycolysis; hepg2 cells; antioxidant defense ... See more keywords