Down-regulation of KCa2.3 channels causes erectile dysfunction in mice
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DOI: 10.1038/s41598-017-04188-5
Abstract: Modulation of endothelial calcium-activated K+ channels has been proposed as an approach to restore arterial endothelial cell function in disease. We hypothesized that small-conductance calcium-activated K+ channels (KCa2.3 or SK3) contributes to erectile function. The… read more here.
Keywords: kca2; kca2 dox; regulation kca2; mice ... See more keywords