Targeting the DNA Damage Response for Cancer Therapy by Inhibiting the Kinase Wee1
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DOI: 10.3389/fonc.2022.828684
Abstract: Cancer cells typically heavily rely on the G2/M checkpoint to survive endogenous and exogenous DNA damage, such as genotoxic stress due to genome instability or radiation and chemotherapy. The key regulator of the G2/M checkpoint,… read more here.
Keywords: dna damage; kinase; wee1; kinase wee1 ... See more keywords