Early-life exposure to the endocrine disruptor 17-α-ethinylestradiol induces delayed effects in adult brain, liver and ovotestis proteomes of a self-fertilizing fish.
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DOI: 10.1016/j.jprot.2018.12.008
Abstract: Early-life represents a critically sensitive window to endocrine disrupting chemicals, potentially leading to long-term repercussions on the phenotype later in life. The mechanisms underlying this phenomenon, referred to as the Developmental Origins of Health and… read more here.
Keywords: brain liver; life; early life; liver ovotestis ... See more keywords