Tenascin C is dysregulated in hypoplastic lungs of miR-200b−/− mice
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DOI: 10.1007/s00383-022-05096-0
Abstract: We previously demonstrated that absence of miR-200b results in abnormal lung development in congenital diaphragmatic hernia due to imbalance between epithelial and mesenchymal cells. Tenascin C is a highly conserved extracellular matrix protein involved in… read more here.
Keywords: tenascin; lung; lungs mir; mir 200b ... See more keywords