MiR‐101a ameliorates AngII‐mediated hypertensive nephropathy by blockade of TGFβ/Smad3 and NF‐κB signalling in a mouse model of hypertension
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DOI: 10.1111/1440-1681.13042
Abstract: Hypertensive nephropathy, clinically characterized by progressive renal fibrosis and inflammation, is a severe complication of hypertension. The objectives of this study were to investigate the roles of miR‐101a in relieving angiotensin II (Ang II)‐mediated hypertensive… read more here.
Keywords: hypertensive nephropathy; mir 101a; mouse model; tgf smad3 ... See more keywords