Acute administration of methionine and/or methionine sulfoxide impairs redox status and induces apoptosis in rat cerebral cortex
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DOI: 10.1007/s11011-017-0054-9
Abstract: High plasma levels of methionine (Met) and its metabolites such as methionine sulfoxide (MetO) may occur in several genetic abnormalities. Patients with hypermethioninemia can present neurological dysfunction; however, the neurotoxicity mechanisms induced by these amino… read more here.
Keywords: cerebral cortex; meto; meto met; met meto ... See more keywords