MFN2 mediates ER-mitochondrial coupling during ER stress through specialized stable contact sites
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DOI: 10.3389/fcell.2022.918691
Abstract: Endoplasmic reticulum (ER) functions critically depend on a suitable ATP supply to fuel ER chaperons and protein trafficking. A disruption of the ability of the ER to traffic and fold proteins leads to ER stress… read more here.
Keywords: stress; atp supply; mitochondrial coupling; mfn2 mediates ... See more keywords