Deletion of Bak1 alleviates microglial necroptosis and neuroinflammation after experimental subarachnoid hemorrhage
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DOI: 10.1111/jnc.15751
Abstract: Microglial necroptosis exacerbates neurodegenerative diseases, central nervous system (CNS) injury, and demonstrates a proinflammatory process, but its contribution to subarachnoid hemorrhage (SAH) is poorly characterized. BCL‐2 homologous antagonist‐killer protein (Bak1), a critical regulatory molecule of… read more here.
Keywords: necroptosis; microglial necroptosis; subarachnoid hemorrhage; bak1 ... See more keywords