Suppression of the RAC1/MLK3/p38 Signaling Pathway by β-Elemene Alleviates Sepsis-Associated Encephalopathy in Mice
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DOI: 10.3389/fnins.2019.00358
Abstract: It is still difficult to treat sepsis-associated encephalopathy (SAE) which is a diffuse brain dysfunction caused by sepsis, with excessive activation of microglia as one of the main mechanisms. Ras-related C3 botulinum toxin substrate 1… read more here.
Keywords: rac1; mlk3 p38; rac1 mlk3; p38 signaling ... See more keywords