TMEM2 Modulates ER Stress in a Non-canonical Manner.
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DOI: 10.1016/j.cmet.2019.11.008
Abstract: Cells utilize multiple mechanisms to support endoplasmic reticulum (ER) function. The unfolded protein response, UPRER, is engaged during proteotoxic challenges to either mitigate ER stress or promote apoptosis. In a CRISPR-based genetic screen, Schinzel et al.… read more here.
Keywords: tmem2 modulates; modulates stress; tmem2; non canonical ... See more keywords