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Published in 2022 at "Cancer Research"
DOI: 10.1158/0008-5472.can-21-3214
Abstract: Loss of RBMS3 cooperates with BRAFV600E to induce lung tumorigenesis, providing a deeper understanding of the molecular mechanisms underlying mutant BRAF-driven lung cancer and potential strategies to more effectively target this disease.
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Keywords:
transposon mutagenesis;
mutagenesis reveals;
driven lung;
lung tumorigenesis ... See more keywords