The Effects of N-terminal Mutations on β-amyloid Peptide Aggregation and Toxicity
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DOI: 10.1016/j.neuroscience.2018.03.014
Abstract: Human amyloid β1-42 (hAβ1-42) peptides are known to self-aggregate into oligomers that contribute to the degeneration of neurons and development of Alzheimer's disease (AD) pathology. Unlike humans, rodents do not develop AD, possibly due to… read more here.
Keywords: effects terminal; mutations amyloid; aggregation toxicity; toxicity ... See more keywords