N2L, a novel lipoic acid-niacin dimer protects HT22 cells against β-amyloid peptide-induced damage through attenuating apoptosis
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DOI: 10.1007/s11011-019-00482-5
Abstract: β-amyloid protein (Aβ) is thought to be the primary cause of the pathogenesis of Alzheimer’s disease (AD). Niacin has been reported to have beneficial effects on AD. Previously, we synthesized a novel compound lipoicacid–niacin dimer… read more here.
Keywords: apoptosis; n2l novel; niacin dimer; ht22 cells ... See more keywords