Ogfod1 deletion increases cardiac beta-alanine levels and protects mice against ischemia-reperfusion injury.
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DOI: 10.1093/cvr/cvab323
Abstract: AIMS Prolyl hydroxylation is a post-translational modification that regulates protein stability, turnover, and activity. The proteins that catalyze prolyl hydroxylation belong to the 2-oxoglutarate- and iron-dependent oxygenase family of proteins. 2-oxoglutarate- and iron-dependent oxygenase domain-containing… read more here.
Keywords: ogfod1 deletion; beta alanine; beta; ischemia reperfusion ... See more keywords