Suppression of PCSK9/NF-kB-dependent pathways by acetate ameliorates cardiac inflammation in a rat model of polycystic ovarian syndrome.
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DOI: 10.1016/j.lfs.2022.120560
Abstract: AIM Endocrinometabolic disorders in women of reproductive age, including polycystic ovarian syndrome (PCOS) has contributed to increased prevalence of cardiovascular disease (CVD) risk and its attendant complications. Acetate, the most abundant endogenously produced short chain… read more here.
Keywords: pcsk9 dependent; pcsk9; polycystic ovarian; rat model ... See more keywords