Liraglutide Treatment Ameliorates Neurotoxicity Induced by Stable Silencing of Pin1
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DOI: 10.3390/ijms20205064
Abstract: Post-translational modulation of peptidylprolyl isomerase Pin1 might link impaired glucose metabolism and neurodegeneration, being Pin1 effectors target for the glucagon-Like-Peptide1 analog liraglutide. We tested the hypotheses in Pin1 silenced cells (SH-SY5Y) treated with 2-deoxy-d-glucose (2DG)… read more here.
Keywords: cell viability; liraglutide treatment; pin1 silencing; treatment ameliorates ... See more keywords