HDAC6 Interacts With Poly (GA) and Modulates its Accumulation in c9FTD/ALS
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DOI: 10.3389/fcell.2021.809942
Abstract: The aberrant translation of a repeat expansion in chromosome 9 open reading frame 72 (C9orf72), the most common cause of frontotemporal dementia (FTD) and amyotrophic lateral sclerosis (ALS), results in the accumulation of toxic dipeptide… read more here.
Keywords: hdac6 interacts; interacts poly; c9ftd als; poly modulates ... See more keywords