Prenatal bisphenol a exposure leads to reproductive hazards on male offspring via the Akt/mTOR and mitochondrial apoptosis pathways
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DOI: 10.1002/tox.22300
Abstract: Bisphenol A (BPA) exposure is ubiquitous, and in laboratory animals and humans, exposure has been associated with male spermatogenesis dysfunction. However, it is largely unknown if this association has a fetal origin. read more here.
Keywords: bisphenol; bisphenol exposure; exposure; leads reproductive ... See more keywords