Prenatal glucocorticoid administration enhances bilirubin metabolic capacity and increases Ugt1a and Abcc2 gene expression via glucocorticoid receptor and PXR in rat fetal liver
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DOI: 10.1111/jog.15235
Abstract: Jaundice is especially common in premature infant born before 35 weeks. Because the premature infant liver is not fully developed at birth it may be incomplete the bilirubin metabolism. The purpose was to evaluate the metabolism… read more here.
Keywords: liver; rat; glucocorticoid administration; prenatal glucocorticoid ... See more keywords