Hyperoxia Causes Mitochondrial Fragmentation in Pulmonary Endothelial Cells by Increasing Expression of Pro-Fission Proteins
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DOI: 10.1161/atvbaha.117.310605
Abstract: Objective— We explored mechanisms that alter mitochondrial structure and function in pulmonary endothelial cells (PEC) function after hyperoxia. Approach and Results— Mitochondrial structures of PECs exposed to hyperoxia or normoxia were visualized and mitochondrial fragmentation… read more here.
Keywords: iii; hyperoxia; expression pro; fragmentation ... See more keywords