Aspirin modulates 2-hydroxyisobutyrylation of ENO1K281 to attenuate the glycolysis and proliferation of hepatoma cells.
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DOI: 10.1016/j.bbrc.2021.04.083
Abstract: Aspirin can efficiently inhibit the glycolysis and proliferation of cancer cells, however, the underlying mechanism is poorly understood. Here, we report that aspirin attenuates the glycolysis and proliferation of hepatoma cells through modulating the levels… read more here.
Keywords: hepatoma cells; glycolysis proliferation; proliferation hepatoma; proliferation ... See more keywords