NETs promote ROS production to induce human amniotic epithelial cell apoptosis via ERK1/2 signaling in spontaneous preterm birth
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DOI: 10.1111/aji.13656
Abstract: Premature birth is a common obstetric complication but its pathogenesis is unclear. Inflammation at the maternal‐fetal interface in preterm labor leads to the infiltration of neutrophils, which promotes inflammatory responses and induces the degradation of… read more here.
Keywords: preterm labor; cell; cell apoptosis; promote ros ... See more keywords