Destruction in maternal-fetal interface of URSA patients via the increase of the HMGB1-RAGE/TLR2/TLR4-NF-κB signaling pathway.
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DOI: 10.1016/j.lfs.2020.117543
Abstract: AIMS HMGB1 has been reported to play a crucial role in the physiological and pathophysiological responses during pregnancy. However, it is still unknown whether excessively expressed HMGB1 at the maternal-fetal interface related to Unexplained Recurrent… read more here.
Keywords: hmgb1; tlr2 tlr4; rage tlr2; maternal fetal ... See more keywords