Up-regulation of ubiquitin-proteasome activity upon loss of NatA-dependent N-terminal acetylation.
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DOI: 10.26508/lsa.202000730
Abstract: N-terminal acetylation is a prominent protein modification, and inactivation of N-terminal acetyltransferases (NATs) cause protein homeostasis stress. Using multiplexed protein stability profiling with linear ubiquitin fusions as reporters for the activity of the ubiquitin proteasome… read more here.
Keywords: acetylation; terminal acetylation; regulation ubiquitin; activity ... See more keywords