Pharmacodynamics and molecular correlates of response to glofitamab in relapsed/refractory non-Hodgkin lymphoma
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DOI: 10.1182/bloodadvances.2021005954
Abstract: Key Points Glofitamab induced dose-dependent induction of cytokines and T-cell margination, proliferation, and activation in peripheral blood. Tumor cell intrinsic factors (eg, TP53 and MYC signaling) are associated with resistance to glofitamab. read more here.
Keywords: pharmacodynamics molecular; molecular correlates; correlates response; glofitamab ... See more keywords