Injured Podocytes Are Sensitized to Angiotensin II-Induced Calcium Signaling.
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DOI: 10.1681/asn.2019020109
Abstract: BACKGROUND Inhibition of angiotensin II (AngII) signaling, a therapeutic mainstay of glomerular kidney diseases, is thought to act primarily through regulating glomerular blood flow and reducing filtration pressure. Although extravascular actions of AngII have been… read more here.
Keywords: induced calcium; responsiveness angii; calcium; injured podocytes ... See more keywords