A detrimental role of RelB in mature oligodendrocytes during experimental acute encephalomyelitis
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DOI: 10.1186/s12974-019-1548-7
Abstract: BackgroundMultiple sclerosis (MS) is an autoimmune demyelinating disease of the central nervous system (CNS). It is firmly established that overactivation of the p65 (RelA) nuclear factor kappa B (NF-κB) transcription factor upregulates expression of inflammatory… read more here.
Keywords: inflammation; cns; relb; role relb ... See more keywords