N-Acetylcysteine, an ROS Inhibitor, Alleviates the Pathophysiology of Hyperthyroidism-Induced Cardiomyopathy via the ROS/Ca2+ Pathway
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DOI: 10.3390/biom12091195
Abstract: Hyperthyroidism is common and can induce cardiomyopathy, but there is no effective therapeutic strategy. The purpose of this study was to investigate the molecular mechanism of hyperthyroidism-induced cardiomyopathy (HTC) and the effect of N-acetylcysteine (NAC),… read more here.
Keywords: hyperthyroidism induced; pathophysiology hyperthyroidism; ros inhibitor; ros ca2 ... See more keywords