TWEAK/Fn14 promotes oxidative stress through AMPK/PGC‑1α/MnSOD signaling pathway in endothelial cells.
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DOI: 10.3892/mmr.2017.8090
Abstract: Tumor necrosis factor-like weak inducer of apoptosis (TWEAK) contributes to dysfunction of endothelial cells via its receptor, Fn14. However, its role in the production of reactive oxygen species (ROS), particularly mitochondrial ROS (mtROS) and the… read more here.
Keywords: ros mtros; tweak fn14; endothelial cells; mnsod ... See more keywords