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   Articles with "ruvbl2" as a keyword



Characterizing the mitochondrial DNA polymerase gamma interactome by BioID identifies Ruvbl2 localizes to the mitochondria.

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recommendations! Published in 2017 at "Mitochondrion"

DOI: 10.1016/j.mito.2016.11.001

Abstract: Human mitochondrial DNA (mtDNA) is replicated by the mitochondrial DNA polymerase gamma (POLG). Using proximity dependent biotin labelling (BioID), we characterized the POLG interactome and identified new interaction partners involved in mtDNA maintenance, transcription, translation… read more here.

Keywords: dna; dna polymerase; polymerase gamma; ruvbl2 ... See more keywords

Identification of RUVBL2 as a novel biomarker to predict the prognosis and drug sensitivity in multiple myeloma based on ferroptosis genes.

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recommendations! Published in 2025 at "Hematology"

DOI: 10.1080/16078454.2025.2467499

Abstract: ABSTRACT Background Multiple myeloma (MM) is a hematological malignancy with the proliferation of malignant plasma cells. Numerous studies have highlighted the critical role of ferroptosis in MM. However, how to use ferroptosis-related genes (FRGs) for… read more here.

Keywords: multiple myeloma; analysis; ruvbl2; prognosis ... See more keywords
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Chemical perturbations reveal that RUVBL2 regulates the circadian phase in mammals

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recommendations! Published in 2020 at "Science Translational Medicine"

DOI: 10.1126/scitranslmed.aba0769

Abstract: A compound that disassembles the circadian repressive super-complex can reset circadian phase and relieve jet lag in mice. Shifting clock gears Molecular circadian oscillators help control sleep/wake periodicity. Ju et al. show that an AAA-type… read more here.

Keywords: phase; circadian phase; ruvbl2; clock ... See more keywords

Ruvbl2 Suppresses Cardiomyocyte Proliferation During Zebrafish Heart Development and Regeneration

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recommendations! Published in 2022 at "Frontiers in Cell and Developmental Biology"

DOI: 10.3389/fcell.2022.800594

Abstract: Cardiomyocyte proliferation is an important source of new myocardium during heart development and regeneration. Consequently, mutations in drivers of cardiomyocyte proliferation cause congenital heart disease, and infarcted human hearts scar because cardiomyocytes exit the cell… read more here.

Keywords: regeneration; heart development; cardiomyocyte proliferation; ruvbl2 ... See more keywords