Selenoprotein N1 Redox Activity Leads to Mitochondrial Dysfunction in Skeletal Muscle
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DOI: 10.1016/j.freeradbiomed.2017.10.312
Abstract: Selenoprotein N (SEPN1), is a broadly expressed resident protein of the endoplasmic reticulum (ER) whose loss of function inexplicably leads to human muscle disease. We have defined the redox-regulated interactome of SEPN1 and identified the… read more here.
Keywords: skeletal muscle; sepn1; redox; selenoprotein ... See more keywords