SHP1 regulates a STAT6–ITGB3 axis in FLT3ITD-positive AML cells
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DOI: 10.1038/s41375-019-0676-5
Abstract: Acute myeloid leukemia (AML) is a life-threatening disease with limited treatment opportunities. FMS-like tyrosine kinase 3 with internal tandem duplications (FLT3ITD) is a constitutively active receptor tyrosine kinase, found in 25–30% of AML cases and… read more here.
Keywords: shp1 depletion; supplementary fig; disease; shp1 ... See more keywords