Interleukin-17A influences the vulnerability rather than the size of established atherosclerotic plaques in apolipoprotein E-deficient mice
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DOI: 10.1515/biol-2022-0072
Abstract: Abstract Interleukin (IL)-17A plays a role in the development of atherosclerotic plaques; however, the mechanism remains unclear. In this study, apolipoprotein E-deficient (ApoE–/–) mice were fed a high-fat diet to induce atherosclerosis, followed by the… read more here.
Keywords: vulnerability; established atherosclerotic; apolipoprotein deficient; size established ... See more keywords