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Published in 2019 at "Frontiers in Physiology"
DOI: 10.3389/fphys.2018.01936
Abstract: When fasted as larvae or fed ketogenic diets as adults, homozygous zebrafish slc16a6a mutants develop hepatic steatosis because their livers cannot export the major ketone body β-hydroxybutyrate, diverting liver-trapped ketogenic carbon atoms to triacylglycerol. Here,…
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Keywords:
length;
monocarboxylate transporter;
slc16a6 regulates;
regulates adult ... See more keywords