Altered gene expression in slc4a11−/− mouse cornea highlights SLC4A11 roles
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DOI: 10.1038/s41598-021-98921-w
Abstract: SLC4A11 is a H+/NH3/water transport protein, of corneal endothelial cells. SLC4A11 mutations cause congenital hereditary endothelial dystrophy and some cases of Fuchs endothelial corneal dystrophy. To probe SLC4A11’s roles, we compared gene expression in RNA… read more here.
Keywords: cornea; gene expression; expression; slc4a11 ... See more keywords