The role of distal tubule and collecting duct sodium reabsorption in sunitinib-induced hypertension
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DOI: 10.1097/hjh.0000000000001650
Abstract: Objective: Antiangiogenic receptor tyrosine kinase inhibitors (RTKI) induce arterial hypertension which may limit their use. Renal fractional sodium excretion (FENa) is reduced in early RTKI-induced hypertension, whereas fractional lithium excretion is unaltered. Therefore, we tested… read more here.
Keywords: enac; induced hypertension; sodium reabsorption; sunitinib ... See more keywords