Targeting hepatic serine-arginine protein kinase 2 ameliorates alcohol-associated liver disease by alternative splicing control of lipogenesis.
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DOI: 10.1097/hep.0000000000000433
Abstract: BACKGROUND AIMS Lipid accumulation induced by alcohol consumption is not only an early pathophysiological response but also a prerequisite for the progression of alcohol-associated liver disease (ALD). Alternative splicing regulates gene expression and protein diversity;… read more here.
Keywords: srpk2; serine arginine; kinase; alternative splicing ... See more keywords