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Published in 2018 at "Brain Research"
DOI: 10.1016/j.brainres.2018.03.023
Abstract: Excessive glutamate efflux and N-methyl-D-aspartate receptor (NMDAR) over-activation represent well-known hallmarks of cerebral ischemia/reperfusion (I/R) injury, still, expression of proteins involved in this aspect of I/R pathophysiology show inconsistent data. Neurosteroid dehydroepiandrosterone (DHEA) has been…
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Keywords:
nmdar;
cerebral ischemia;
male wistar;
synaptic nmdar ... See more keywords